We seek postdoctoral candidate with an interest in pulmonary vascular signaling, cell biology, and cell metabolism to study the molecular and cellular mechanisms of pulmonary vascular remodeling as it relates to pulmonary hypertension with specific focus on cell signaling, metabolism, and cell-cell interactions in regulating pulmonary vascular remodeling. The successful applicant should have expertise in cellular and molecular biology techniques (ie. western blotting, cell transfections/ infections, proliferation and apoptosis assays). Expertise in cell-cell interactions, cellular metabolism, cell culture techniques and small animal (rats and mice) models of pulmonary hypertension is appreciated.
To be considered for the position, please send in a detailed CV with methodological expertise, a list of scientific publications, and the contact details of three referees to email@example.com. Salary will be according to NIH scale and commensurate with experience. Position available immediately.
Goncharova lab research interests have focused on the molecular and cellular mechanisms of pulmonary arterial hypertension (PAH). We use molecular and cellular techniques, human tissues and cells, transgenic mice, and rodent models of pulmonary hypertension to dissect new molecular signaling hubs driving hyper-proliferation of resident pulmonary vascular cells, pulmonary vascular remodeling and PAH and to identify and pre-clinically validate novel molecular target pathways for therapeutic intervention. Ongoing projects are focused on the cross-talk of mTOR and HIPPO signaling networks in pulmonary vascular cell remodeling and right ventricular dysfunction in PAH; the role of GATA6 transcriptional factor as a regulator of pulmonary vascular cell-cell communications, BMP signaling and inflammation in PAH; and role of post-translational modifications in pulmonary vascular remodeling.
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